We've been seeing a lot of decompensated liver disease recently, likely related to increases in alcohol intake due to the COVID pandemic. Over the next month, several chapters on critical care hepatology be released, exploring how to manage these patients. We start with hepatorenal syndrome and hepatorenal physiology, because this is often the crux of how and why these patients devolve into multi-organ failure.
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The IBCC chapter is located 👉 here.
- The podcast & comments are below.
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Hi
I don’t see the podcast!! Just I tune logo
Hi Josh
I am embarrassed to say that after many years I never knew what this entity is until now having read your thorough, clear, simple, concise description in your IBCC chapter. it’s a little tricky, but not terribly hard to grasp. excellent chapter Josh. thank you
Hey there, thanks for the great article!
You uploaded the wrong pdf (it is about heatstroke)
Hello Dr. Farkas,
Why is in your opinion the following treatment approach would not work:
High Ag II causing vasoconstriction of efferent arteriole to the point of proximal tubules hypoxia>>>give Ace inh. which can prevent the conversion of Ag I to Ag II and effectively decrease Ag II level >> relax efferent art and reverse hypoxia…
Sincerely,
Aleksei
Excuse me, Can I ask why in hypervolemia 25% albumin is better ?I think it will cause overload???
What do you think about this recently published (CHEST) and high-quality (GRADE methodology) revision of albumin use? https://pubmed.ncbi.nlm.nih.gov/38447639/ In HRS, no RCTs. In cirrosis in general, we had the negative ATTIRE Prevention of PICD is based on surrogates improvement (as far as I remember when I reviewed it, the improvement was lower serum creatinine after some days After PBS seems the only one with patiented-oriented outcomes improvement including mortality, but even here the 2 largest RCTs are confliting I think albumin (in general or in hepatic disease) could be a theme for a PulmCrit blog or even a Wee with… Read more »