ALGORITHM🫀
- Rhythm
- Intervals
- Axis
- Chambers
- Morphology
Index🫀
- 2° block; 3° block
- AFib
- Aflutter
- AIVR
- Alternans, electrical
- Aneurysm, anterior
- Anterior OMI
- Artifact
- ARVD
- Ashmann phenomenon
- Atria:
- AVNRT
- Bayes syndrome
- BER (benign early repolarization)
- Benign TWI
- Biatrial abnormality
- Bidirectional VT
- Biventricular hypertrophy
- Bradycardia
- Brugada
- Clumped beating
- DCM
- De Winter T-waves
- Dextrocardia
- Digoxin
- Electrical alternans
- Emphysema pattern
- ER (early repolarization)
- Escape rhythms
- Extreme axis deviation
- FAT
- Fragmentation of QRS
- HCM
- Heart block
- 1st degree
- 2nd degree (I, II)
- 3rd degree
- High lead placement of V1 & V2
- HYPER
- HYPO
- Inferior OMI
- Irregularity:
- Bradycardia (including clumped beats)
- Tachycardia
- J–waves
- Junctional
- Juvenile TWI
- LAA
- LAD (left axis deviation)
- LAHB
- Lateral OMI
- LBBB iLBBB
- Lead I sign
- Lead misplacement
- Low Voltage
- LPFB
- Lung hyperinflation pattern
- LVH
- MAT
- Mobitz
- Myocarditis
- Narrow Complex Tach
- North-west axis
- OMI
- Osborne waves
- P-wave abnormalities
- PAC
- Paced ECG analysis
- PE
- Pectus excavatum
- Pericardial effusion
- Pericarditis
- Persistent S-wave in V6
- PJC
- Pneumothorax, left
- Polymorphic VT
- Poor R-wave progression
- Posterior OMI
- POTS
- PR interval:
- Premature beats
- Prominent T-waves
- PRWP
- PVC
- Q–waves
- QT interval:
- R-wave transition:
- R-V1 is tall
- RAA
- RAD (right axis deviation)
- RBBB; partial RBBB
- Renal failure
- Retained S-wave in V6
- rSr' in V1
- RVH
- RVMI w/ IMI; isolated RVMI
- S-I is prominent
- Shark fin
- Sinus node dysfunction
- Sinus node reentrant tachycardia
- Sinus tachycardia
- Speed limits for tachycardia
- Sodium channel blocker
- South African Flag sign
- STD
- STE
- Stress cardiomyopathy
- SVT
- T-Wave
- Tachycardia:
- Takotsubo cardiomyopathy
- Tall R-wave in V1
- TdP
- TTV1
- Torsade de pointes
- TWI
- U-waves
- Ventricular escape
- Ventricular parasystole
- WAP
- Wellens
- Wide complex
- WPW
[1] rhythm & rate
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rhythm
- Look at every beat in the rhythm strip to confirm that they all are consistent with your rhythm diagnosis.
- Don't settle for a diagnosis that doesn't truly explain the ECG.
rate
- 300 – 150 – 100
- 75 – 60 – 50
- 43 – 37 – 33
(related)
[2a] PR interval
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PR <120 ms (3 boxes) 📖
- Tachycardia with short PR (flutter, atrial tach with 2:1 block, AVRT, JT).
- With inverted P-waves, consider:
- Low ectopic atrial rhythm.
- Junctional rhythm.
- WPW.
- Benign (asymptomatic, PR ~100-110).
PR >5 200 ms (5 boxes)
- 1st degree AV block. 📖
- Normally, PR decreases with tachycardia (so borderline PR may indicate 1st degree block in the context of tachycardia).
[2b] QRS >120 ms? 📖
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ddx of QRS >120 ms
- Abnormal P-waves:
- Hyperkalemia
- Hypothermia
- MI with pseudo-wide QRS
- RBBB or Na-blocker 🚀
- LBBB or LVH 🚀
- IVCD
- Pacemaker
ddx of QRS >200 ms
- Hyperkalemia.
- Acidosis.
- Sodium channel blocker toxicity.
- Shark-fin (STE with hyperacute T-wave that seems to widen the QRS complex).
- Ventricular tachycardia.
[2c] QT interval
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⚠️ Be sure to look for the longest QT interval among various ECG leads.
long QT (>1/2 RR if HR<100) 📖
- ? QRS prolongation.
- ? Long ST segment: hypocalcemia, hypothermia, LQTS3.
- ? T-wave with double-hump morphology: Hypokalemia, medications that inhibit the IKr channel, LQTS2.
- ? Hypothermia.
- ? Takotsubo CM.
- ? Myocarditis.
short QT 📖
- QT <360 ms or 9 boxes.
- QTc <~350 ms.
[3a] vertical axis & abnormal S-I
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LAD (negative aVF and II) 🚀
- Prior IMI.
- LAHB, criteria: 📖
- [1] QRS <120 ms (often ~100-120 ms).
- [2] Frontal axis between -45° to -90°:
- Positive in Lead I.
- Negative in aVF.
- Substantially net negative in Lead II.
- S-wave in aVF > R-wave in Lead I.
- [3] qR in aVL.
- [4] R-peak time in aVL of ≧45 ms.
- LVH.
RAD 📖
- S-I prominent (terminal RAD)
- S-I >1-1.5 mm is often regarded as abnormal.
- S-I > R-I is definitely abnormal.
- Coexisting S-wave in V6 supports abnormality.
[3b] P-wave axis
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normal P-wave axis is 0° to +75°:
- P-wave should be upright in I and II. (aVF will usually be positive but could be mute if the P-wave axis is close to 0°).
- P-wave should be inverted in aVR.
rightward P-axis deviation (>75 degrees)
- P-wave becomes negative in aVL.
- Causes include:
- RAA (right atrial enlargement).
- Vertical heart configuration (e.g., emphysema without RAA).
- Ectopic atrial rhythm or conduction abnormality.
leftward P-axis deviation (<0 degrees)
- P-wave becomes negative in aVF.
- Causes include:
- LAA (left atrial enlargement).
- Horizontal heart configuration (e.g., morbid obesity).
- Ectopic atrial rhythm or conduction abnormality.
[3c] horizontal axis
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- Tall R-wave in V1
- R-wave > S-wave.
- Poor R-wave progression (>V4)
- Persistent S-wave in V6
[4a] atria
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LAA 📖
- [1] V1 negative deflection >40 ms x 1 box. However, consider high lead placement if: 📖
- V2 with negative/biphasic P-wave and/or TWI.
- QRS in aVR looks similar to V1.
- Rsr' in V1 and/or V2, often with saddleback STE.
- [2] Broad P-wave in inferior leads (e.g., P-mitrale).
- 💡 Look carefully for LVH or BiV hypertrophy.
RAA 📖
- [1] >1.5 mm P-wave in V1 or V2 (insensitive).
- [2] P-pulmonale (peaked & >2.5 mm in lead II): causes
[4b] LVH 📖
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voltage criteria for LVH
Romhilt-Estes (4 high prob; ≧5 def)
- Voltage criteria (3 points): any
- R or S in limb leads ≧20 mm.
- S in V1 or V2 ≧30 mm.
- R in V5 or V6 ≧30 mm.
- LV strain pattern: 3 points (1 with digoxin).
- LAA in V1 (terminal P-wave is >40ms and >1 mm): 3 points.
- LAD: 2 points.
- QRS >90 ms: 1 point.
- Delayed intrinsicoid deflection in V5 or V6 (>50 ms): 1 point.
BiV hypertrophy
- RVH features often include:
- RAA.
- Terminal RAD (S-I).
- (R-V1 plus S-V5/6) > 10.5 mm.
- LVH features often include:
- LAA.
- High voltages (including R or S in limb leads ≧20 mm).
- Katz-Wachtel: >~50 mm biphasic (R~S) QRS amplitude in V2-V4.
[4c] low voltage 📖
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definition of low voltage
- Whole QRS <5 mm in all limb leads.
- Whole QRS <10 mm in all precordial leads.
(links)
[5a] pathological Q-waves 📖
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1
[5b] other morphological abnormalities
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reminders & general principles for clinical ECG analysis
- Always be systematic.
- Avoid satisfaction of search (multiple problems often coexist & superimpose).
- Identify and name specific ECG findings (e.g., tall RV1, diffuse STD with STE in aVR).
- Include differential diagnoses rather than attempting to discern a single diagnosis.
- Avoid relying on subjective impressions of morphology.
- When in doubt, tend to err towards the more dangerous/acute diagnosis (“worst first”).
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